IRS2 (Insulin receptor substrate 2) variants and mutations

IRS2 (also known as Insulin receptor substrate 2) is a human protein-coding gene encoding an insulin receptor substrate 2 protein. It relays insulin and growth-factor signals in liver, pancreatic beta cells, brain, and other tissues, supporting metabolic control and cell survival. Altered signaling can contribute to insulin resistance and diabetes, but strong monogenic human disease associations are limited. This analysis covers 3,604 IRS2 variants and mutations. Of these, 73% have computational variant effect predictions. Disease context includes neurodegenerative disease, type 2 diabetes mellitus, and prostate carcinoma. Example IRS2 variants include S3G, S3N, and P4L.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, AlphaFold DB, gnomAD constraint, Open Targets, MaveDB, LitVar.

Notable IRS2 variants

Examples include S3G, S3N, P4L, P4Q, P4S, P5R, R6L, R6P. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.