LPA (Apolipoprotein(a)) variants and mutations

LPA (also known as Apolipoprotein(a)) is a human protein-coding gene encoding an apolipoprotein(a) protein. Its apolipoprotein(a) component covalently attaches to an LDL-like particle to form lipoprotein(a), whose circulating level is largely genetically determined. High Lp(a) is a causal risk factor for atherosclerotic cardiovascular disease and calcific aortic-valve disease. This analysis covers 2,741 LPA variants and mutations. Of these, 75% have computational variant effect predictions. Disease context includes cardiovascular disorder, coronary artery disorder, and myocardial infarction. Example LPA variants include M1?, H3L, and H3Y.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, AlphaFold DB, gnomAD constraint, Open Targets, MaveDB, LitVar.

Notable LPA variants

Examples include M1?, H3L, H3Y, K4*, K4E, K4R, E5K, E5W. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.