REN (Renin) variants and mutations

REN (also known as Renin) is a human protein-coding gene encoding a renin protein. It initiates the renin-angiotensin cascade by cleaving angiotensinogen, thereby controlling blood pressure, sodium balance, and extracellular fluid volume. Pathogenic variants can cause renal tubular dysgenesis or rare inherited tubulointerstitial kidney disease depending on their effect on renin production. This analysis covers 627 REN variants and mutations. Of these, 92% have computational variant effect predictions. Disease context includes familial juvenile hyperuricemic nephropathy type 2, renal tubular dysgenesis, and renal tubular dysgenesis of genetic origin. Example REN variants include G3E, G3V, and R5G.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable REN variants

Examples include G3E, G3V, R5G, R6K, R6S, M7I, M7L, P8A. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.