KRAS (GTPase KRas) variants and mutations

KRAS (also known as GTPase KRas) is a human protein-coding gene encoding a GTPase protein. A small GTPase that acts as a molecular switch in the RAS-MAPK signaling pathway. By cycling between GDP- and GTP-bound states, it relays growth and survival signals, and activating KRAS variants are common drivers of cancer. This analysis covers 825 KRAS variants and mutations. Of these, 41% have computational variant effect predictions. Disease context includes Noonan syndrome, Noonan syndrome 3, and cardiofaciocutaneous syndrome 2. Example KRAS variants include M1*, T2P, and T2S.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable KRAS variants

Examples include M1*, T2P, T2S, E3D, E3K, E3G, Y4*, Y4F. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.