LEP (Leptin) variants and mutations

LEP (also known as Leptin) is a human protein-coding gene encoding a leptin protein. It signals nutritional energy stores to the hypothalamus and suppresses appetite while supporting normal endocrine and immune function. Biallelic loss-of-function variants cause severe early-onset obesity with hyperphagia and can impair pubertal development and immunity. This analysis covers 356 LEP variants and mutations. Of these, 82% have computational variant effect predictions. Disease context includes obesity due to congenital leptin deficiency, type 2 diabetes mellitus, and spondylolisthesis. Example LEP variants include H2R, H2H, and W3C.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, MaveDB, LitVar.

Notable LEP variants

Examples include H2R, H2H, W3C, W3R, W3*, G4*, G4G, T5N. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.