LRRK2 (Q5S007) variants and mutations

LRRK2 (also known as Q5S007) is a human protein-coding gene encoding a leucine-rich repeat serine/threonine-protein kinase 2 protein. Its kinase and GTPase activities regulate Rab proteins, vesicle trafficking, lysosomal function, and cellular stress responses. Gain-of-function variants, especially G2019S, are among the most common genetic causes of autosomal dominant Parkinson disease. This analysis covers 4,051 LRRK2 variants and mutations. Of these, 81% have computational variant effect predictions. Disease context includes Hereditary late-onset Parkinson disease, Parkinson disease, and Young adult-onset Parkinsonism. Example LRRK2 variants include M1?, A2G, and A2V.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, MaveDB, LitVar.

Notable LRRK2 variants

Examples include M1?, A2G, A2V, S3T, S3S, G4A, G4D, G4G. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.