TCF7L2 (Transcription factor 7-like 2) variants and mutations

TCF7L2 (also known as Transcription factor 7-like 2) is a human protein-coding gene encoding a transcription factor 7-like 2 protein. It mediates Wnt-dependent transcription and also influences pancreatic, hepatic, and intestinal programs involved in glucose metabolism. Common intronic variants at this locus are among the strongest and most reproducible genetic risk factors for type 2 diabetes. This analysis covers 2,522 TCF7L2 variants and mutations. Of these, 26% have computational variant effect predictions. Disease context includes type 2 diabetes mellitus, colorectal adenocarcinoma, and prostate carcinoma. Example TCF7L2 variants include M1?, P2A, and P2L.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, Interaction Network Analysis, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable TCF7L2 variants

Examples include M1?, P2A, P2L, P2R, P2S, P2T, P2Q, P2P. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.