TP53 (Cellular tumor antigen p53) variants and mutations

TP53 (also known as Cellular tumor antigen p53) is a human protein-coding gene encoding a cellular tumor antigen p53 protein. It coordinates transcriptional responses to DNA damage and other cellular stresses, promoting cell-cycle arrest, senescence, DNA repair, or apoptosis when appropriate. Loss of this tumor-suppressive control is one of the most common events in cancer, while germline pathogenic variants cause Li-Fraumeni syndrome. This analysis covers 2,743 TP53 variants and mutations. Of these, 89% have computational variant effect predictions. Disease context includes Li-Fraumeni syndrome, hepatocellular carcinoma, and head and neck squamous cell carcinoma. Example TP53 variants include M1?, E2*, and E2D.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, PharmGKB, MaveDB, LitVar.

Notable TP53 variants

Examples include M1?, E2*, E2D, E2G, E2K, E2Q, E3*, E3G. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.