F11 (Coagulation factor XI) variants and mutations

F11 (also known as Coagulation factor XI) is a human protein-coding gene encoding a coagulation factor XI protein. Its activated form amplifies thrombin generation through the intrinsic coagulation pathway. Deficiency causes hemophilia C with variable bleeding, while reduced factor XI activity is associated with lower thrombosis risk and is being explored as a safer anticoagulation target. This analysis covers 1,261 F11 variants and mutations. Of these, 60% have computational variant effect predictions. Disease context includes neurodegenerative disease, thrombophilia, and Familial exudative vitreoretinopathy. Example F11 variants include M1I, M1K, and L4*.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, MaveDB, LitVar.

Notable F11 variants

Examples include M1I, M1K, L4*, Y5*, Y5C, Q6*, Q6H, Q6R. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.