DNMT1 (P26358) variants and mutations

DNMT1 (also known as P26358) is a human protein-coding gene encoding a DNA (cytosine-5)-methyltransferase 1 protein. It copies existing DNA methylation patterns during replication and also contributes to chromatin regulation and neuronal maintenance. Dominant pathogenic variants can cause hereditary sensory neuropathy with dementia and hearing loss or a cerebellar ataxia-deafness-narcolepsy syndrome. This analysis covers 1,968 DNMT1 variants and mutations. Of these, 40% have computational variant effect predictions. Disease context includes autosomal dominant cerebellar ataxia, deafness and narcolepsy, hereditary sensory neuropathy-deafness-dementia syndrome, and acute myeloid leukemia. Example DNMT1 variants include M1?, P2A, and P2L.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, MaveDB, LitVar.

Notable DNMT1 variants

Examples include M1?, P2A, P2L, P2R, P2S, A3V, A6T, P7A. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.