EGFR (Epidermal growth factor receptor) variants and mutations

EGFR (also known as Epidermal growth factor receptor) is a human protein-coding gene encoding an epidermal growth factor receptor protein. A cell-surface receptor tyrosine kinase that responds to epidermal-growth-factor family ligands. Ligand binding activates phosphorylation cascades that regulate cell growth, survival, and differentiation, which is why EGFR changes are important in cancer biology. This analysis covers 5,559 EGFR variants and mutations. Of these, 48% have computational variant effect predictions. Disease context includes non-small cell lung carcinoma, lung adenocarcinoma, and cancer. Example EGFR variants include R2*, R2G, and R2L.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable EGFR variants

Examples include R2*, R2G, R2L, R2P, R2Q, R2R, P3A, P3H. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.