APOB (Apolipoprotein B-100) variants and mutations

APOB (also known as Apolipoprotein B-100) is a human protein-coding gene encoding an apolipoprotein B-100 protein. It provides the structural backbone for triglyceride-rich lipoproteins and LDL, while ApoB-100 also mediates LDL-receptor binding and clearance. Pathogenic variants can cause familial hypobetalipoproteinemia or defective ApoB-related hypercholesterolemia depending on their effect on particle assembly and receptor binding. This analysis covers 7,169 APOB variants and mutations. Of these, 76% have computational variant effect predictions. Disease context includes hypercholesterolemia, autosomal dominant, type B, Hypercholesterolemia, and familial hypobetalipoproteinemia 1. Example APOB variants include M1?, D2A, and D2G.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable APOB variants

Examples include M1?, D2A, D2G, D2N, P3L, P3Q, P4L, P4R. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.