F5 (Coagulation factor V) variants and mutations

F5 (also known as Coagulation factor V) is a human protein-coding gene encoding a coagulation factor V protein. After activation, it serves as an essential cofactor for factor Xa in the prothrombinase complex and greatly accelerates thrombin generation. Deficiency can cause bleeding, whereas factor V Leiden produces activated-protein-C resistance and substantially increases venous-thrombosis risk. This analysis covers 2,967 F5 variants and mutations. Of these, 86% have computational variant effect predictions. Disease context includes thrombophilia due to activated protein C resistance, congenital factor V deficiency, and factor V deficiency. Example F5 variants include F2L, G4A, and G4S.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable F5 variants

Examples include F2L, G4A, G4S, C5G, R7C, R7H, L8F, W9R. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.