STAT3 (P40763) variants and mutations

STAT3 (also known as P40763) is a human protein-coding gene encoding a signal transducer and activator of transcription 3 protein. It translates cytokine and growth-factor signals into transcriptional programs governing immune regulation, survival, proliferation, and tissue repair. Dominant-negative variants cause hyper-IgE syndrome, while activating germline variants cause early autoimmunity and lymphoproliferation and somatic activation contributes to cancer. This analysis covers 1,620 STAT3 variants and mutations. Of these, 36% have computational variant effect predictions. Disease context includes Autosomal dominant hyper-IgE syndrome, autoimmune disease, and hyper-IgE recurrent infection syndrome 1, autosomal dominant. Example STAT3 variants include A2S, A2T, and A2V.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable STAT3 variants

Examples include A2S, A2T, A2V, Q3H, W4*, N5S, N5Y, Q6E. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.