PLP1 (Myelin proteolipid protein) variants and mutations

PLP1 (also known as Myelin proteolipid protein) is a human protein-coding gene encoding a myelin proteolipid protein. It supports central-nervous-system myelin structure and is required for oligodendrocyte and axonal integrity. Gene duplication most commonly causes Pelizaeus-Merzbacher disease, while other variants can cause spastic paraplegia type 2 or milder leukodystrophy. This analysis covers 564 PLP1 variants and mutations. Of these, 63% have computational variant effect predictions. Disease context includes Pelizeaus-Merzbacher spectrum disorder, hereditary spastic paraplegia 2, and Spastic paraplegia type 2. Example PLP1 variants include M1?, M1I, and M1K.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable PLP1 variants

Examples include M1?, M1I, M1K, M1R, M1T, M1V, G2D, G2R. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.