NRAS (GTPase NRas) variants and mutations

NRAS (also known as GTPase NRas) is a human protein-coding gene encoding a GTPase protein. Its active GTP-bound state drives RAF-MEK-ERK and PI3K signaling downstream of growth-factor receptors. Somatic activating variants are common drivers of melanoma, leukemia, and other cancers, while germline activating variants can cause Noonan syndrome. This analysis covers 800 NRAS variants and mutations. Of these, 60% have computational variant effect predictions. Disease context includes Noonan syndrome, Noonan syndrome 6, and large congenital melanocytic nevus. Example NRAS variants include p.Met1dup, T2A, and T2I.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable NRAS variants

Examples include p.Met1dup, T2A, T2I, T2S, T2N, E3*, E3A, E3D. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.