ITGA2B (Integrin alpha-IIb) variants and mutations

ITGA2B (also known as Integrin alpha-IIb) is a human protein-coding gene encoding an integrin alpha-IIb protein. Together with ITGB3, it forms the major platelet fibrinogen receptor that becomes activated during platelet stimulation and drives aggregation. Biallelic loss-of-function variants cause Glanzmann thrombasthenia, a severe inherited platelet-aggregation disorder. This analysis covers 1,569 ITGA2B variants and mutations. Of these, 81% have computational variant effect predictions. Disease context includes Glanzmann thrombasthenia 1, Glanzmann thrombasthenia, and autosomal dominant macrothrombocytopenia. Example ITGA2B variants include M1?, A2D, and R3G.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable ITGA2B variants

Examples include M1?, A2D, R3G, R3K, R3S, A4S, A4T, C6Y. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.