APC (Adenomatous polyposis coli protein) variants and mutations

APC (also known as Adenomatous polyposis coli protein) is a human protein-coding gene encoding an adenomatous polyposis coli protein. A tumor-suppressor protein that promotes the removal of beta-catenin and helps keep Wnt signaling under control. It also organizes microtubules and actin in the cell, and inherited APC disruption is strongly associated with familial adenomatous polyposis and colorectal tumor risk. This analysis covers 14,982 APC variants and mutations. Of these, 93% have computational variant effect predictions. Disease context includes familial adenomatous polyposis 1, Familial adenomatous polyposis, and Desmoid-type fibromatosis. Example APC variants include M1I, A2D, and A2G.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, gnomAD constraint, Open Targets, ClinGen, PharmGKB, MaveDB, LitVar.

Notable APC variants

Examples include M1I, A2D, A2G, A2P, A2S, A2T, A2V, A3E. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.