SOS2 (Son of sevenless homolog 2) variants and mutations

SOS2 (also known as Son of sevenless homolog 2) is a human protein-coding gene encoding a son of sevenless homolog 2 protein. It activates RAS by catalyzing GDP-GTP exchange downstream of growth-factor receptors. Germline activating variants cause Noonan syndrome, generally through increased RAS-MAPK signaling. This analysis covers 1,818 SOS2 variants and mutations. Of these, 88% have computational variant effect predictions. Disease context includes Noonan syndrome, Noonan syndrome 9, and hypertensive disorder. Example SOS2 variants include M1T, Q2L, and A4S.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable SOS2 variants

Examples include M1T, Q2L, A4S, P5A, Q6*, P7R, P7S, P7T. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.