RAG1 (P15918) variants and mutations

RAG1 (also known as P15918) is a human protein-coding gene encoding a v(D)J recombination-activating protein 1 protein. It initiates V(D)J recombination by cutting antigen-receptor gene segments, creating the enormous receptor diversity required for adaptive immunity. Biallelic severe loss-of-function variants cause severe combined immunodeficiency, while hypomorphic alleles can cause Omenn syndrome or combined immunodeficiency with autoimmunity. This analysis covers 1,794 RAG1 variants and mutations. Of these, 83% have computational variant effect predictions. Disease context includes Omenn syndrome, severe combined immunodeficiency, autosomal recessive, T cell-negative, B cell-n, and combined immunodeficiency with skin granulomas. Example RAG1 variants include M1V, A2T, and A2V.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable RAG1 variants

Examples include M1V, A2T, A2V, A3V, A3P, A3A, S4P, S4T. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.