LOX (Protein-lysine 6-oxidase) variants and mutations

LOX (also known as Protein-lysine 6-oxidase) is a human protein-coding gene encoding a protein-lysine 6-oxidase protein. It oxidatively initiates covalent crosslinking of collagen and elastin, strengthening arteries and other extracellular matrices. Pathogenic loss-of-function variants can weaken the aortic wall and predispose to familial thoracic aortic aneurysm and dissection. This analysis covers 781 LOX variants and mutations. Of these, 75% have computational variant effect predictions. Disease context includes familial thoracic aortic aneurysm and aortic dissection, Abnormality of the cardiovascular system, and Aortic dissection. Example LOX variants include R2C, R2S, and A4P.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, AlphaFold DB, gnomAD constraint, Open Targets, MaveDB, LitVar.

Notable LOX variants

Examples include R2C, R2S, A4P, A4T, W5*, W5R, W5S, T6I. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.