PTEN (Phosphatidylinositol 3,4,5-trisphosphate 3-phosphatase and dual-) variants and mutations

PTEN (also known as Phosphatidylinositol 3,4,5-trisphosphate 3-phosphatase and dual-) is a human protein-coding gene encoding a phosphatidylinositol 3,4,5-trisphosphate 3-phosphatase and dual-specificity protein phosphatase protein. A lipid and protein phosphatase that removes phosphate groups from signaling molecules, especially PIP3. By opposing the PI3K-AKT pathway, it limits cell growth and survival signals, and PTEN variants are associated with Cowden syndrome and multiple cancers. This analysis covers 2,275 PTEN variants and mutations. Of these, 49% have computational variant effect predictions. Disease context includes Cowden syndrome 1, macrocephaly-autism syndrome, and PTEN hamartoma tumor syndrome. Example PTEN variants include M1?, M1L, and T2A.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable PTEN variants

Examples include M1?, M1L, T2A, T2I, T2K, T2R, T2S, A3D. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.