MET (P08581) variants and mutations

MET (also known as P08581) is a human protein-coding gene encoding a hepatocyte growth factor receptor protein. Hepatocyte-growth-factor signaling through this pathway promotes cell survival, proliferation, motility, and invasive growth during development and tissue repair. Exon 14 skipping, amplification, activating mutations, or fusions can drive cancer and create actionable therapeutic dependencies. This analysis covers 6,533 MET variants and mutations. Of these, 40% have computational variant effect predictions. Disease context includes papillary renal cell carcinoma, hereditary papillary renal cell carcinoma, and hepatocellular carcinoma. Example MET variants include M1?, K2*, and K2E.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable MET variants

Examples include M1?, K2*, K2E, K2N, K2R, K2K, A3D, A3G. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.