C5 (Complement C5) variants and mutations

C5 (also known as Complement C5) is a human protein-coding gene encoding a complement protein. Its cleavage produces the potent inflammatory mediator C5a and C5b, which initiates assembly of the membrane-attack complex. Excessive activation contributes to several complement-mediated disorders, making C5 blockade an established therapeutic strategy. This analysis covers 1,883 C5 variants and mutations. Of these, 92% have computational variant effect predictions. Disease context includes Immunodeficiency due to a late component of complements deficiency, paroxysmal nocturnal hemoglobinuria, and myasthenia gravis. Example C5 variants include M1?, G2D, and G2S.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, MaveDB, LitVar.

Notable C5 variants

Examples include M1?, G2D, G2S, L3V, L4F, G5E, I6L, I6R. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.