DDC (P20711) variants and mutations

DDC (also known as P20711) is a human protein-coding gene encoding an aromatic-L-amino-acid decarboxylase protein. It converts L-DOPA to dopamine and 5-hydroxytryptophan to serotonin, making it essential for monoamine neurotransmitter synthesis. Biallelic loss-of-function variants cause aromatic L-amino-acid decarboxylase deficiency with severe movement and autonomic abnormalities. This analysis covers 903 DDC variants and mutations. Of these, 70% have computational variant effect predictions. Disease context includes aromatic L-amino acid decarboxylase deficiency, Parkinson disease, and hereditary disease. Example DDC variants include M1?, N2I, and A3T.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, MaveDB, LitVar.

Notable DDC variants

Examples include M1?, N2I, A3T, S4I, S4R, S4T, F6L, R7*. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.