RASA1 (Ras GTPase-activating protein 1) variants and mutations

RASA1 (also known as Ras GTPase-activating protein 1) is a human protein-coding gene encoding a ras GTPase-activating protein 1 protein. It accelerates hydrolysis of RAS-GTP and therefore limits RAS-MAPK signaling downstream of growth-factor receptors. Haploinsufficiency causes capillary malformation-arteriovenous malformation syndrome and related fast-flow vascular anomalies. This analysis covers 1,535 RASA1 variants and mutations. Of these, 67% have computational variant effect predictions. Disease context includes capillary malformation-arteriovenous malformation 1, Capillary malformation - arteriovenous malformation, and capillary malformation-arteriovenous malformation syndrome. Example RASA1 variants include M1?, M2I, and A3T.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable RASA1 variants

Examples include M1?, M2I, A3T, A3V, A3E, A3A, A4G, A4T. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.