ALK (ALK tyrosine kinase receptor) variants and mutations

ALK (also known as ALK tyrosine kinase receptor) is a human protein-coding gene encoding an ALK tyrosine kinase receptor protein. Its kinase signaling influences neural development and cell growth, but constitutive activation can become strongly oncogenic. ALK fusions, activating mutations, or amplification drive anaplastic large-cell lymphoma, subsets of lung cancer, and neuroblastoma and can be targeted with ALK inhibitors. This analysis covers 6,292 ALK variants and mutations. Of these, 61% have computational variant effect predictions. Disease context includes neuroblastoma, neuroblastoma, susceptibility to, 3, and non-small cell lung carcinoma. Example ALK variants include M1L, M1T, and G2*.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable ALK variants

Examples include M1L, M1T, G2*, G2A, G2E, G2R, G2V, A3G. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.