UMOD (Uromodulin) variants and mutations

UMOD (also known as Uromodulin) is a human protein-coding gene encoding an uromodulin protein. It is secreted by thick-ascending-limb cells into urine, where it contributes to salt handling, urinary defense, and protection against kidney stones. Dominant pathogenic variants cause autosomal dominant tubulointerstitial kidney disease, while common regulatory variants influence kidney-function and hypertension risk. This analysis covers 1,323 UMOD variants and mutations. Of these, 71% have computational variant effect predictions. Disease context includes familial juvenile hyperuricemic nephropathy type 1, autosomal dominant medullary cystic kidney disease with or without hyperuricemia, and chronic kidney disease. Example UMOD variants include M1?, G2V, and Q3*.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, MaveDB, LitVar.

Notable UMOD variants

Examples include M1?, G2V, Q3*, P4Q, P4S, P4T, S5T, L6P. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.