TPM1 (Tropomyosin alpha-1 chain) variants and mutations

TPM1 (also known as Tropomyosin alpha-1 chain) is a human protein-coding gene encoding a tropomyosin alpha-1 chain protein. It lies along actin filaments and helps control access of myosin to actin in response to troponin and calcium, while also stabilizing cytoskeletal actin in nonmuscle cells. Pathogenic variants can cause hypertrophic or dilated cardiomyopathy and several congenital myopathies. This analysis covers 541 TPM1 variants and mutations. Of these, 83% have computational variant effect predictions. Disease context includes hypertrophic cardiomyopathy, left ventricular noncompaction, and left ventricular noncompaction 9. Example TPM1 variants include M1?, D2H, and D2Y.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable TPM1 variants

Examples include M1?, D2H, D2Y, D2D, I4V, I4I, I4M, K5K. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.