TGFBR2 (TGF-beta receptor type-2) variants and mutations

TGFBR2 (also known as TGF-beta receptor type-2) is a human protein-coding gene encoding a TGF-beta receptor type-2 protein. It binds TGF-beta ligands and activates TGFBR1 to initiate canonical and noncanonical signaling. Germline pathogenic variants cause Loeys-Dietz syndrome type 2, while somatic loss can remove growth-suppressive TGF-beta responses in cancer. This analysis covers 2,235 TGFBR2 variants and mutations. Of these, 43% have computational variant effect predictions. Disease context includes Loeys-Dietz syndrome, Loeys-Dietz syndrome 2, and Marfan syndrome. Example TGFBR2 variants include M1?, M1V, and G2C.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable TGFBR2 variants

Examples include M1?, M1V, G2C, G2S, G2D, G2V, G2G, R3P. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.