RELA (Transcription factor p65) variants and mutations

RELA (also known as Transcription factor p65) is a human protein-coding gene encoding a transcription factor p65 protein. It supplies the p65 transcriptional subunit of canonical NF-kappaB, driving inflammatory, immune, survival, and stress-response genes after receptor activation. Haploinsufficiency can cause chronic mucocutaneous ulceration and immune dysregulation, while excessive activation is common in inflammatory disease and cancer. This analysis covers 977 RELA variants and mutations. Of these, 82% have computational variant effect predictions. Disease context includes mucocutaneous ulceration, chronic, neurodegenerative disease, and Alzheimer disease. Example RELA variants include E3*, L4V, and P6S.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, MaveDB, LitVar.

Notable RELA variants

Examples include E3*, L4V, P6S, L7F, I8M, F9V, P10A, P10L. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.