RARA (Retinoic acid receptor alpha) variants and mutations

RARA (also known as Retinoic acid receptor alpha) is a human protein-coding gene encoding a retinoic acid receptor alpha protein. It converts retinoic-acid binding into transcriptional programs that promote differentiation and development. The PML::RARA fusion blocks myeloid maturation in acute promyelocytic leukemia, a mechanism directly reversed by all-trans retinoic acid and arsenic-based therapy. This analysis covers 1,148 RARA variants and mutations. Of these, 48% have computational variant effect predictions. Disease context includes acne, acute promyelocytic leukemia, and psoriasis. Example RARA variants include M1?, M1V, and M1T.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, MaveDB, LitVar.

Notable RARA variants

Examples include M1?, M1V, M1T, M1I, M1L, A2T, A2S, A2V. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.