LMNA (Prelamin-A/C) variants and mutations

LMNA (also known as Prelamin-A/C) is a human protein-coding gene encoding a prelamin-A/C protein. The gene product produces lamins A and C, structural proteins that form the nuclear lamina beneath the inner nuclear membrane. Lamins help maintain nuclear shape and organize chromatin, and LMNA variants are associated with muscular dystrophy, cardiomyopathy, lipodystrophy, and premature-aging syndromes. This analysis covers 1,553 LMNA variants and mutations. Of these, 95% have computational variant effect predictions. Disease context includes dilated cardiomyopathy, Emery-Dreifuss muscular dystrophy 2, autosomal dominant, and familial partial lipodystrophy, Dunnigan type. Example LMNA variants include M1I, M1K, and M1L.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, PharmGKB, MaveDB, LitVar.

Notable LMNA variants

Examples include M1I, M1K, M1L, M1V, E2*, E2G, E2K, E2Q. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.