KCNA2 (P16389) variants and mutations

KCNA2 (also known as P16389) is a human protein-coding gene encoding a potassium voltage-gated channel subfamily A member 2 protein. Its current helps repolarize neuronal membranes and regulate action-potential firing and neurotransmitter release. Both loss- and gain-of-function variants can cause developmental and epileptic encephalopathy, often with ataxia, movement abnormalities, or intellectual disability. This analysis covers 1,013 KCNA2 variants and mutations. Of these, 71% have computational variant effect predictions. Disease context includes undetermined early-onset epileptic encephalopathy, genetic developmental and epileptic encephalopathy, and multiple sclerosis. Example KCNA2 variants include M1?, A4V, and T5N.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable KCNA2 variants

Examples include M1?, A4V, T5N, G6R, D7A, D7N, D7V, P8L. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.