F9 (Coagulation factor IX) variants and mutations

F9 (also known as Coagulation factor IX) is a human protein-coding gene encoding a coagulation factor IX protein. Its activated form combines with factor VIIIa to efficiently activate factor X during coagulation. Pathogenic loss-of-function variants cause X-linked hemophilia B, with bleeding severity determined largely by residual factor IX activity. This analysis covers 905 F9 variants and mutations. Of these, 62% have computational variant effect predictions. Disease context includes hemophilia B, hemophilia A, and thrombophilia, X-linked, due to factor 9 defect. Example F9 variants include M1?, Q2L, and Q2P.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable F9 variants

Examples include M1?, Q2L, Q2P, Q2R, R3C, R3H, R3S, R3P. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.