SCN9A (Q15858) variants and mutations

SCN9A (also known as Q15858) is a human protein-coding gene encoding a sodium channel protein type 9 subunit alpha protein. The protein forms Nav1.7, a voltage-gated sodium channel that amplifies electrical signals in peripheral sensory neurons. Changes in Nav1.7 activity can produce either excessive pain or congenital insensitivity to pain, making SCN9A central to pain biology. This analysis covers 4,096 SCN9A variants and mutations. Of these, 76% have computational variant effect predictions. Disease context includes primary erythermalgia, paroxysmal extreme pain disorder, and channelopathy-associated congenital insensitivity to pain, autosomal recessive. Example SCN9A variants include M1?, A2S, and A2T.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, MaveDB, LitVar.

Notable SCN9A variants

Examples include M1?, A2S, A2T, A2V, M3I, M3K, M3L, L4*. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.