RAD51C (O43502) variants and mutations

RAD51C (also known as O43502) is a human protein-coding gene encoding a DNA repair protein RAD51 homolog 3 protein. It participates in RAD51-paralog complexes that promote homologous-recombination repair and restart damaged replication forks. Heterozygous loss-of-function variants increase ovarian and breast-cancer risk, while biallelic variants can cause Fanconi anemia. This analysis covers 1,846 RAD51C variants and mutations. Of these, 62% have computational variant effect predictions. Disease context includes Hereditary breast and ovarian cancer syndrome, Fanconi anemia complementation group O, and Fanconi anemia. Example RAD51C variants include M1I, M1L, and M1T.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable RAD51C variants

Examples include M1I, M1L, M1T, M1V, R2C, R2G, R2H, R2P. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.