NPHS2 (Podocin) variants and mutations

NPHS2 (also known as Podocin) is a human protein-coding gene encoding a podocin protein. It organizes nephrin-containing slit-diaphragm complexes in podocytes and helps maintain the glomerular filtration barrier. Biallelic pathogenic variants are a major cause of steroid-resistant nephrotic syndrome and focal segmental glomerulosclerosis. This analysis covers 763 NPHS2 variants and mutations. Of these, 79% have computational variant effect predictions. Disease context includes nephrotic syndrome, nephrotic syndrome, type 2, and familial idiopathic steroid-resistant nephrotic syndrome. Example NPHS2 variants include M1I, M1L, and E2D.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, AlphaFold DB, gnomAD constraint, Open Targets, MaveDB, LitVar.

Notable NPHS2 variants

Examples include M1I, M1L, E2D, R3G, R3K, R3S, R4K, A5E. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.