NF1 (Neurofibromin) variants and mutations

NF1 (also known as Neurofibromin) is a human protein-coding gene encoding a neurofibromin protein. It accelerates conversion of active RAS-GTP to inactive RAS-GDP and therefore restrains RAS-MAPK signaling. Loss-of-function variants cause neurofibromatosis type 1 with neurofibromas, pigmentary features, learning difficulties, and increased tumor risk. This analysis covers 13,101 NF1 variants and mutations. Of these, 54% have computational variant effect predictions. Disease context includes neurofibromatosis type 1, neurofibromatosis-Noonan syndrome, and neurofibromatosis. Example NF1 variants include M1?, M1I, and M1K.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable NF1 variants

Examples include M1?, M1I, M1K, M1L, M1R, M1T, M1V, A2D. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.