MYPN (Myopalladin) variants and mutations

MYPN (also known as Myopalladin) is a human protein-coding gene encoding a myopalladin protein. It links sarcomeric and cytoskeletal proteins at the Z-disc and intercalated-disc regions, helping transmit mechanical force and maintain muscle architecture. Pathogenic variants can cause dilated or hypertrophic cardiomyopathy and, in some cases, skeletal myopathy. This analysis covers 2,299 MYPN variants and mutations. Of these, 67% have computational variant effect predictions. Disease context includes dilated cardiomyopathy 1KK, MYPN-related myopathy, and Abnormality of the cardiovascular system. Example MYPN variants include M1T, M1V, and Q2R.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable MYPN variants

Examples include M1T, M1V, Q2R, Q2*, D3H, D3D, D4G, D4N. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.