JUP (Junction plakoglobin) variants and mutations

JUP (also known as Junction plakoglobin) is a human protein-coding gene encoding a junction plakoglobin protein. It links desmosomal and adherens-junction cadherins to the cytoskeleton and helps maintain mechanical coupling between cardiomyocytes and epithelial cells. Pathogenic variants can cause arrhythmogenic cardiomyopathy and cardiocutaneous syndromes such as Naxos disease. This analysis covers 1,314 JUP variants and mutations. Of these, 75% have computational variant effect predictions. Disease context includes Naxos disease, arrhythmogenic right ventricular dysplasia 12, and Arrhythmogenic right ventricular dysplasia. Example JUP variants include E2D, E2G, and M4I.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, MaveDB, LitVar.

Notable JUP variants

Examples include E2D, E2G, M4I, M4V, M7I, M7T, Q9*, Q9E. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.