FOXP3 (Forkhead box protein P3) variants and mutations

FOXP3 (also known as Forkhead box protein P3) is a human protein-coding gene encoding a forkhead box protein P3 protein. It establishes the transcriptional program of regulatory T cells and is essential for maintaining peripheral immune tolerance. Loss-of-function variants cause IPEX syndrome with severe early-onset autoimmunity, enteropathy, eczema, and endocrine disease. This analysis covers 610 FOXP3 variants and mutations. Of these, 81% have computational variant effect predictions. Disease context includes immune dysregulation-polyendocrinopathy-enteropathy-X-linked syndrome, type 1 diabetes mellitus, and hydrops fetalis. Example FOXP3 variants include M1I, M1K, and N3I.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, MaveDB, LitVar.

Notable FOXP3 variants

Examples include M1I, M1K, N3I, N3K, P4S, R5G, R5M, G7A. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.