FLNC (Filamin-C) variants and mutations

FLNC (also known as Filamin-C) is a human protein-coding gene encoding a filamin-C protein. It crosslinks actin and anchors signaling and structural proteins at Z-discs, costameres, and other mechanically stressed sites in striated muscle. Pathogenic variants can cause arrhythmogenic or dilated cardiomyopathy as well as myofibrillar and distal myopathies. This analysis covers 4,379 FLNC variants and mutations. Of these, 73% have computational variant effect predictions. Disease context includes hypertrophic cardiomyopathy 26, myofibrillar myopathy 5, and distal myopathy with posterior leg and anterior hand involvement. Example FLNC variants include M2T, M2V, and M2L.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable FLNC variants

Examples include M2T, M2V, M2L, M2R, M2I, N4S, S5G, S5R. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.