DNM1 (Dynamin-1) variants and mutations

DNM1 (also known as Dynamin-1) is a human protein-coding gene encoding a dynamin-1 protein. It drives membrane fission during synaptic-vesicle endocytosis, allowing rapid recycling of vesicles after neurotransmitter release. De novo pathogenic variants can cause severe developmental and epileptic encephalopathy with profound developmental impairment. This analysis covers 1,076 DNM1 variants and mutations. Of these, 77% have computational variant effect predictions. Disease context includes Lennox-Gastaut syndrome, developmental and epileptic encephalopathy, 31A, and developmental and epileptic encephalopathy, 31B. Example DNM1 variants include M1I, M1T, and G2D.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable DNM1 variants

Examples include M1I, M1T, G2D, G2S, G2C, G2V, G2A, G2G. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.