CSF3R (Q99062) variants and mutations

CSF3R (also known as Q99062) is a human protein-coding gene encoding a granulocyte colony-stimulating factor receptor protein. It transmits G-CSF signals that promote neutrophil precursor proliferation, differentiation, and survival. Activating or truncating somatic variants are major drivers of chronic neutrophilic leukemia, while loss-of-function variants can cause severe congenital neutropenia. This analysis covers 1,611 CSF3R variants and mutations. Of these, 68% have computational variant effect predictions. Disease context includes autosomal recessive severe congenital neutropenia due to CSF3R deficiency, myelodysplastic syndrome, and neoplasm. Example CSF3R variants include M1?, A2T, and A2V.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, MaveDB, LitVar.

Notable CSF3R variants

Examples include M1?, A2T, A2V, R3S, L4P, L4V, G5A, G5E. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.