Joubert syndrome: genes and variants

Joubert syndrome is linked to 3 analyzed proteins (CEP290, SUFU and CHD7). 7 DNA variants are known to cause it; 850 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Also known as: Joubert syndrome 1; Joubert syndrome 10; Joubert syndrome 32; Joubert syndrome 5

Genes linked to Joubert syndrome

Weakly linked (only a few uncertain records): IFT172 and PKD1.

Where Joubert syndrome variants cluster

Known disease-causing variants in Joubert syndrome

VariantPositionProtein partClinical label
CEP290 M1I1Self-association (with itself or C-terminus)Disease-causing (★★)
CEP290 M1V1Self-association (with itself or C-terminus)Disease-causing (★★)
CHD7 R2319C2319Disease-causing (★★)
CEP290 E1568D1568Coiled coilDisease-causing (★)
CEP290 E1572K1572Coiled coilDisease-causing (★)
CEP290 I5T5Self-association (with itself or C-terminus)Disease-causing (★)
SUFU H176R176Disease-causing

Same protein, different disease

Diseases related to Joubert syndrome

Frequently asked questions

Which genes are linked to Joubert syndrome?

In CATVariant, Joubert syndrome is linked to 3 analyzed proteins: CEP290 (Centrosomal protein of 290 kDa), SUFU (Suppressor of fused homolog) and CHD7 (ATP-dependent chromatin remodeler CHD7).

How many genetic variants are linked to Joubert syndrome?

903 variants: 7 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 850 are of uncertain significance or have conflicting reports.

Which uncertain variants in Joubert syndrome look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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