CHD7 (Q9P2D1) variants and mutations

CHD7 (also known as Q9P2D1) is a human protein-coding gene encoding an ATP-dependent chromatin remodeler protein. It regulates chromatin accessibility and developmental gene programs across multiple embryonic tissues. Haploinsufficiency is the major cause of CHARGE syndrome, which can affect the eyes, heart, choanae, growth, genital development, ears, and nervous system. This analysis covers 4,212 CHD7 variants and mutations. Of these, 83% have computational variant effect predictions. Disease context includes CHARGE syndrome, hypogonadotropic hypogonadism 5 with or without anosmia, and CHD7-related CHARGE syndrome. Example CHD7 variants include A2E, A2G, and A2T.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable CHD7 variants

Examples include A2E, A2G, A2T, A2V, A2A, D3N, D3E, P4L. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.