G6PD deficiency: genes and variants

G6PD deficiency is linked to 1 analyzed protein (G6PD). 11 DNA variants are known to cause it; 4 more are uncertain, and 1 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Genes linked to G6PD deficiency

Known disease-causing variants in G6PD deficiency

VariantPositionProtein partClinical label
G6PD R227W227Disease-causing (★★)
G6PD P50S50Disease-causing (★★)
G6PD R198H198Disease-causing (★★)
G6PD P353S353Disease-causing (★★)
G6PD R459P459Disease-causing (★★)
G6PD V68M68Disease-causing (★★)
G6PD F173L173Disease-causing (★★)
G6PD R459G459Disease-causing (★★)
G6PD R463S463Disease-causing (★★)
G6PD R227L227Disease-causing
G6PD R257L257Disease-causing

Uncertain variants in G6PD deficiency that look disease-causing

VariantPositionProtein partClinical labelEvidence
G6PD R227Q227Conflicting reports (★)+6: 2 other pathogenic changes within 3 positions; R227W at the same position is pathogenic; REVEL 0.869

Same protein, different disease

Diseases related to G6PD deficiency

Frequently asked questions

Which genes are linked to G6PD deficiency?

In CATVariant, G6PD deficiency is linked to 1 analyzed protein: G6PD (Glucose-6-phosphate 1-dehydrogenase).

How many genetic variants are linked to G6PD deficiency?

32 variants: 11 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 4 are of uncertain significance or have conflicting reports.

Which uncertain variants in G6PD deficiency look disease-causing?

1 uncertain variants reach the likely-pathogenic range of the ACMG/AMP points scale on computable evidence, for example G6PD R227Q. These are leads for expert review, not diagnoses.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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