G6PD (P11413) variants and mutations

G6PD (also known as P11413) is a human protein-coding gene encoding a glucose-6-phosphate 1-dehydrogenase protein. It generates NADPH through the pentose-phosphate pathway, providing red blood cells with the reducing power needed to withstand oxidative stress. Deficiency can trigger neonatal jaundice or acute hemolytic anemia after infections, fava beans, or oxidant drugs. This analysis covers 869 G6PD variants and mutations. Of these, 67% have computational variant effect predictions. Disease context includes anemia, nonspherocytic hemolytic, due to G6PD deficiency, anemia (phenotype), and G6PD deficiency. Example G6PD variants include M1?, E3K, and E3Q.

Variant analysis overview

Variant and mutation evidence

Clinical, disease, and population context

Protein structure and variant hotspots

Data sources

Evidence in this analysis draws on EBI Proteins Variation, UniProt, gnomAD v4, EuropePMC, 3D Hotspot Analysis, Interaction Network Analysis, Protein Data Bank, AlphaFold DB, gnomAD constraint, Open Targets, ClinGen, MaveDB, LitVar.

Notable G6PD variants

Examples include M1?, E3K, E3Q, Q4K, Q4R, A6V, S8C, S8I. Listed records include available protein-change notation, database identifiers, clinical classifications, computational predictions, population evidence, experimental measurements, and disease context.