Familial Mediterranean fever: genes and variants

Familial Mediterranean fever is linked to 5 analyzed proteins (MEFV, IL1B, TUBA1A, TUBB2B and TUBB3). 6 DNA variants are known to cause it; 424 more are uncertain, and 0 of those already look disease-causing on computable evidence.

Last updated 2026-09-30. Research information, not medical advice.

Also known as: Familial Mediterranean fever, autosomal dominant

Genes linked to Familial Mediterranean fever

Weakly linked (only a few uncertain records): MPL.

Where Familial Mediterranean fever variants cluster

Known disease-causing variants in Familial Mediterranean fever

VariantPositionProtein partClinical label
MEFV S242R242Disease-causing (★★)
MEFV E167D167Disease-causing (★★)
MEFV F479L479Required for homotrimerization and induction of Disease-causing (★★)
MEFV M680I680B30.2/SPRYDisease-causing (★★)
MEFV V726A726B30.2/SPRYDisease-causing (★★)
MEFV R761H761B30.2/SPRYDisease-causing (★★)

Diseases related to Familial Mediterranean fever

Frequently asked questions

Which genes are linked to Familial Mediterranean fever?

In CATVariant, Familial Mediterranean fever is linked to 5 analyzed proteins: MEFV (Pyrin), IL1B (Interleukin-1 beta), TUBA1A (Tubulin alpha-1A chain), TUBB2B (Tubulin beta-2B chain) and TUBB3 (Tubulin beta-3 chain).

How many genetic variants are linked to Familial Mediterranean fever?

479 variants: 6 are classified as disease-causing (pathogenic or likely pathogenic) in ClinVar and 424 are of uncertain significance or have conflicting reports.

Which uncertain variants in Familial Mediterranean fever look disease-causing?

None of the uncertain variants currently reaches the likely-pathogenic range on computable evidence alone.

About this data

Variant–disease links come from ClinVar, Open Targets and UniProt, pooled from the latest public CATVariant analysis of each human protein. Evidence scores use the ACMG/AMP Bayesian points scale with computable criteria only (position among known disease variants, rarity in gnomAD, calibrated predictors, deep mutational scanning); there is no family or patient data, so they prioritise variants for expert review and never classify them.

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